Decrypting Diabetes [Part I]

July 18, 2018July 18, 2018 David Dempsey1 Comment

What is diabetes?

High blood sugar. We test for it by monitoring blood sugar after a fasting period. If your blood sugar is over a certain amount, we call you diabetic.

Symptoms:

Increased thirst
Frequent urination
Extreme hunger
Unexplained weight loss
Fatigue
Irritability
Blurred vision
Slow-healing sores
Frequent infections, such as gums or skin infections and vaginal infections

Type 1 diabetes can develop at any age, though it often appears during childhood or adolescence. Type 2 diabetes, the more common type, can develop at any age, though it’s more common in people older than 40.

But let’s think about this for a second with our other theories in mind. We’ve shown how your brain perceives time, and proven personal relativity. So we know that diabetes is a negative thing, and it’s closely correlated to high blood pressure and heart disease. So people with diabetes have more brain entropy and experience time more slowly, and age faster. But everyone generally eats about the same. If we’re talking meal times. The diabetic mind, seems to store blood sugar. Almost being designed for closer to a fasting environment.

How does blood sugar look over time?

Why would the body release sugars more slowly in people who experience time so slowly? It seems to be counter intuitive.

It’s the same reason that you say old people’s metabolisms slow down. When your body is under stress, time is dilating, and your hunger may be uncontrollable. Think about it, if every hour is an eternity, it’s really not that crazy to be eating that often. The truth is not that drastic, but that’s the idea.

Here’s what it is: blood sugar is external stress. We know that those individuals that age faster heal slower. And those that sunburn faster, recover slower. So naturally, if blood sugar is a stress, the further you are from your ground state, the longer it takes for your body to recover or, in this case, process it.

Why are diabetics so hungry and thirsty, and pee so often?

That’s simple. They perceive time differently. Their time is shortened because of the stress on their bodies and their brains. Just imagine twice the amount of time passing in between meals for you. That’s what it feels like for them.

Can you explain why diabetics would lose weight?

Yes. Here’s how that works: the diabetic continues their eating habits even though their perception of time has changed. Essentially, he or she could be experiencing three days of personal time in a single day, depending on their level of entropy. So if this is true, it all makes sense. The weight loss is explainable.

Why do old people develop it so often?

The entropy of their brains is higher. They are furthest from their ground state. This is why they recover slower, and why they lose their vision.

Is it not just a disease for fat people?

No. It’s way more complicated than that. Plenty of skinny people have it, and plenty of overweight and obese people don’t. I wish it was that simple. It’s just not.

So is it reversible? I think so. I need to dig in to case studies. I know that you can change your perception of time. I guess it depends on how open minded you are. We know the ground state is attainable. The question is ‘what are you willing to do to get there?’

Sources

Rethinking HIV

July 10, 2018July 10, 2018 David Dempsey

HIV is not an sexually-transmitted disease. It’s just not.

How did I come to this conclusion? The better question is how science came to the conclusion that it was.

What is HIV?

HIV stands for human immunodeficiency virus. It is the virus that can lead to acquired immunodeficiency syndrome, or AIDS, if not treated. Unlike some other viruses, the human body can’t get rid of HIV completely, even with treatment. So once you get HIV, you have it for life.

HIV attacks the body’s immune system, specifically the CD4 cells (T cells), which help the immune system fight off infections. Untreated, HIV reduces the number of CD4 cells (T cells) in the body, making the person more likely to get other infections or infection-related cancers. Over time, HIV can destroy so many of these cells that the body can’t fight off infections and disease. These opportunistic infections or cancers take advantage of a very weak immune system and signal that the person has AIDS, the last stage of HIV infection.

How do we diagnose it?

We look at your blood and test for antigens or antibodies. There are all sorts of tests, but my main takeaways: just because you tested negative doesn’t mean you don’t have HIV. Also just because you tested positive doesn’t mean that you have HIV.

Who gets HIV?

Prisoners. In 2010, over 20,000 prisoners had HIV.
Drug Addicts. People who inject drugs are 28 times more likely to get HIV.
Heterosexual Females. The made up 19% of the new cases in 2016.
Gay males. Gay men account for 70% of the new infections in the US.
People in southern Africa. Far and away the most prevalent place for HIV.

Ok. So all of these people either have anal sex or drug addictions? Maybe you can buy into that. I’m going to keep asking questions.

Are there any other things that all these groups have in common?

Yes. Malnourishment. These groups all have different reasons for being malnourished, but they all are. Or can be.

How would you explain the disparity between the black people getting HIV so disproportionally compared to other races?

A couple of different things. First off, they have lower body fat percentages than these other races. You know this already, but this study spells it out. That’s really it. I was going to say socio-economic disparities would be the other thing, but in this day and age it has to be drastic to cause malnourishment. Like in Africa.

What is so unique about Africa that people get HIV so often?

It’s a perfect storm of black people with low body fat and malnourishment. The economic and agricultural climate make it much more difficult to get and stay healthy, whatever that means.

So how do you explain the newborns with HIV in Africa?

They’re born to HIV positive mothers. Those mothers are obviously passing along their nourishment to the child. I don’t think there’s a logical leap here. If the mother is malnourished, the children are likely to be malnourished. Here’s a study looking at the weights of these new borns.

What about drug addicts?

You’ve seen these people. They do not look well. Many of these drugs are appetite suppressants, and they are often forced to choose between their next meal and their next fix.

But straight females get it too, right?

Think about it. Straight females are most likely to have a negative body image or suffer from anorexia or bulimia. People with anorexia may develop immune deficiencies that may alter T-cell populations. That’s essentially what we call HIV.

Why do gay black guys get it so often way more often than any other subset?

A couple different reasons here: they start off with lower body fat percentages. They have malnourishment and/or body image problems. The gay culture is typically one of chiseled jawlines and thin waists. Maybe that’s a stereotype. [Here’s an article that digs into that part of gay culture.] Also worth noting, the receivers are much more likely to get HIV.

Why do old people get it more often?

We’ve covered my opinion on aging, so almost by definition now, the aging population is underweight or malnourished [see How to age like white people]. Because of this they are more vulnerable to immunity problems. 45% of Americans living with HIV were 50 or older. 27% were 55+, and 6% were 65+. In general, older people are more likely to get it. Here’s an article that discusses it in more detail.

Before you call total bullshit, I didn’t invent anything here. I used the data already collected over years of scientific research, and questioned the logic behind it. I am not saying that HIV/AIDS is not real. I am just saying that the cause of the lowered immune response should be questioned. It’s hard to cure a disease when you don’t know what causes it.

Sources:

Cancer is a brain disease

July 4, 2018 David Dempsey

How can I possibly come to this conclusion? It’s pretty straightforward if you’ve read my other stuff. [I’ll link some important source articles below]

Entropy increases in the brain as we age.

Entropy causes aging.

Cancer is the last stage of cellular life. Therefore aging causes cancer.

Aging starts in the brain.

Thus, cancer starts in the brain.

Before you call bullshit, think about the entire field of epigenetics, the study of how your genetic code mutates over time. We know that you weren’t born with cancer. We know that your genetic code changes over time. And we also know that the chances of getting cancer greatly increases as you age.

So, in my opinion, there’s never going to be a magic pill or vaccination to cure cancer. We have to fight the disease at it’s source: the brain. How do we do that? We combat the aging process best we can: Sleep better, see better, and get in that cardio. We measure our personal time dilation, and think critically about the medicines we’re taking, because side effects matter. Take matters into your own hands, because you’ve been controlling things all along anyways, without even knowing it.

Huntington’s is not genetic

June 21, 2018June 21, 2018 David Dempsey

Even doctors admit that it’s hard to distinguish between Huntington’s, Parkinson’s, and Alzheimer’s. Here’s an article saying that treatment for one of these diseases may work for the other two.

Some researchers and physicians consider the differentiation between cortical and sub-cortical dementia important for patient diagnosis, but others remain skeptical that a significant difference exists. The major criticism of the studies that show variation between cortical and sub-cortical dementias is that there is pathological overlap between the sample groups that are used to model the two categories. These studies often assume that Alzheimer’s patients mostly have cortical dementia and HD or Parkinson’s patients preferentially exhibit subcortical dementia. Necropsies have shown, however, that the brains of both Alzheimer’s and HD patients exhibit a certain degree of both categories of dementia.

If in fact both cortical and subcortical dementia occur in Alzheimer’s, HD, and Parkinson’s patients, then these studies may be problematic. As a result, physicians are still trying to learn more about the differences between the pathologies of the diseases in hopes of finding a more reliable way of differentiating dementias. The ability to differentiate dementias may lead researchers and physicians to better diagnose and treat neurodegenerative diseases. [Source]

We’re going to go through the symptoms, but if you’ve been reading, you know how this goes.

So first, here are the symptoms:

Cognitive: amnesia, delusion, lack of concentration, memory loss, mental confusion, slowness in activity, or difficulty thinking and understanding

Muscular: abnormality walking, increased muscle activity, involuntary movements, problems with coordination, loss of muscle, or muscle spasms

Behavioral: compulsive behavior, fidgeting, irritability, or lack of restraint

Psychological: delirium, depression, hallucination, or paranoia

Mood: anxiety, apathy, or mood swings

Also common: tremor, weight loss, or impaired voice

Those look familiar. Are there any symptoms that aren’t covered between Alzheimer’s, Parkinson’s, and schizophrenia?

“For most diseases, symptoms will vary from person to person. People with the same disease may not have all the symptoms listed.” Wait…what? I thought the symptoms were the only things separating this from the other diseases?

And is it really genetic? It’s complicated, but the consensus is yes. Even though 10% of cases are “due to a new mutation.” But what about that field of epigenetics that basically says that your genes can change over time? Here’s the unabridged version.

HD is typically inherited from a person’s parents, although up to 10% of cases are due to a new mutation. The disease is caused by an autosomal dominant mutation in either of an individual’s two copies of a gene called Huntingtin. This means a child of an affected person typically has a 50% chance of inheriting the disease. The Huntingtin gene provides the genetic information for a protein that is also called “huntingtin”. Expansion of CAG (cytosine-adenine-guanine) triplet repeats in the gene coding for the Huntingtin protein results in an abnormal protein, which gradually damages cells in the brain, through mechanisms that are not fully understood. Diagnosis is by genetic testing, which can be carried out at any time, regardless of whether or not symptoms are present. This fact raises several ethical debates: the age at which an individual is considered mature enough to choose testing; whether parents have the right to have their children tested; and managing confidentiality and disclosure of test results.

This segment is made up of a series of three DNA building blocks (cytosine, adenine, and guanine) that appear multiple times in a row. Normally, the CAG segment is repeated 10 to 35 times within the gene. In people with Huntington disease, the CAG segment is repeated 36 to more than 120 times. People with 36 to 39 CAG repeats may or may not develop the signs and symptoms of Huntington disease, while people with 40 or more repeats almost always develop the disorder.

So if you have 27-39 repeats of this code you may or may not get the disease. But if you have 40 or more repeats, you almost always get the disorder. Wait…almost always? So you’re saying even the hard science isn’t foolproof.

The number of CAG repeats in an HD gene can be unstable when the gene is passed on to the next generation. That means the number of CAG repeats can increase or decrease when the gene is passed from parent to child. Wait, it varies from generation to generation? In the sole aspect that we’re using to call it genetic?

Older fathers are more likely to pass along the extended copy of this gene. We’ve talked about aging parents several times before. We know that the age of mothers closely correlates to Down Syndrome, while the age of fathers closely correlates to Dwarfism.

I have a question for you. How often do we test people with dementia for this CAG repeat? I’m guessing there is not much reason to test for Huntington’s when there is no family history.

So in summary, here’s why I don’t think Huntington’s disease is genetic:

10% of cases are “random” mutations
Even the hard science is not absolute
It gets more probable with aging dad’s
Epigenetics. Our genes change over time.

So if it is not genetic, then it is practically indistinguishable from Alzheimers and Parkinson’s.

Did science get it wrong? Maybe so. I think that a bunch of guys spent their lives studying batches of symptoms, it was the least we could do to name these batches of symptoms after them. All the other fields of science do it. Here’s the problem, the more classifications did not lead to more knowledge in this case. Because the symptoms are indistinguishable.

Sources:

High blood pressure starts in the brain

June 19, 2018June 19, 2018 David Dempsey2 Comments

We’ve proven how the brain perceives time, and your blood pressure is a sign of this perception. How? The second derivative of time is a variable in pressure. And this pressure is in the closed system of your body.

Here’s some more detail for those who want it, from a physics nerd. Pressure equals force/area. Force equals mass times acceleration. Acceleration is the change in velocity over time. Time here is relative to the subject. Relative to their brain activity.

We control our own time. Our time is represented in our blood pressure. So current blood pressure essentially equals current time perception. Thus, it would make sense that people with a history of high blood pressure would die the soonest. They are aging the fastest. Well, in theory. One blood pressure reading is really just the instantaneous time perception.

Have I done an adequate job getting to this point? Probably not. We experience time differently. We age differently. These things are related. Athletes age slowest, and use their brains the best. The more stress we have in our lives, the more we age, the faster our time accelerates, and the higher our blood pressure, and the worse athletes we become.

So next time you go to the doctor, and read 140/80, they may be right that you’re going to die early, but they have no idea why. How can the medications they give you solve your problem if they don’t understand the organ that’s effected first?

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Tourette’s is reversible

June 17, 2018June 17, 2018 David Dempsey

We’ll get there in a second. But first, is Tourette’s genetic? The majority of cases are inherited. Kinda. Here’s what Wikipedia says about it:

A person with Tourette’s has about a 50% chance of passing the gene(s) to one of his or her children, but Tourette’s is a condition of variable expression and incomplete penetrance. Thus, not everyone who inherits the genetic vulnerability will show symptoms; even close family members may show different severities of symptoms, or no symptoms at all. The gene(s) may express as Tourette’s, as a milder tic disorder (provisional or chronic tics), or as obsessive–compulsive symptoms without tics. Only a minority of the children who inherit the gene(s) have symptoms severe enough to require medical attention. Gender appears to have a role in the expression of the genetic vulnerability: males are more likely than females to express tics.

I have a science background, and looking at this type of explanation makes me cringe. Read it for yourself, but it seems like we’re trying to make a genetic model fit when it’s really something we just can’t explain.

It’s associated with OCD, ADD, ADHD, and sleep disorders. This shouldn’t surprise you at this point. We’ve discussed the nature of these diseases and how they are more related than anyone knows. In my opinion, the brain is binary. It’s either working properly or it’s not. And if it’s not, there’s a host of different functions that will be impaired. [see There is only one brain disease]

It’s 3-4 times more likely in guys than girls. It’s the same reason that men are more likely to get skin cancer and on average live about six years less. Why? Because of their brain activity. They are further from their ground state, so time feels slower. Recovery takes longer.

White kids are twice as likely as black and Hispanic kids to get Tourette’s. Why is that? Resting brain activity. It’s the same reason that black kids are more likely to play in the NFL. The closer their brains are to the delta state the faster they recover and better they operate.

Tics may remit with age. This is from Wikipedia. But it’s huge. Why? Because using my model of the human brain, this means that Tourette’s is a reversible condition. How do we reverse it? Same way we reverse other brain dysfunctions:

Identify the loop. This is the repeated behavior. It should be pretty obvious.
Identify the fears causing the loop. What are they afraid of? What makes them act like this?
Identify the logic causing the fears. Why are they scared of this?
Doubt the logic. Question their reasoning. If you can change their mind, you can change their brain.

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Dads cause Dwarfism

June 15, 2018June 16, 2018 David Dempsey1 Comment

OK. So a couple days ago we went through Down Syndrome, and how it is closely tied with the age of mothers, and how that means that we play a large role in causing or preventing it.

Today, I want to talk about the dads. We know that there are some strong correlations between the age of dads and a list of birth defects. We need to know a couple things: are they genetic disorders? Is it the lifestyle of the dads that determines these outcomes? Is it predetermined at conception? Or is it something that can be corrected?

What birth defects are closely correlated to the age of their fathers:

autism
schizophrenia
dwarfism/Achondroplasia
Apert’s Syndrome

We’ve already talked about autism and schizophrenia, and how I think they are both curable and why. So while they are equally important, we’re going to focus on dwarfism today.

B9781455727940000085_f008-003-9781455727940 — Mother’s Age And Down Syndrome [left] and Father’s age and Achondroplasia and Apert’s Syndrome [right] Source

It’s a random gene mutation. But it’s random and we don’t even really know why it mutates. Apparently it’s called epigenetics, which essentially undermines genetics, in my opinion. Think about it for a second, the entire field of genetics is about mapping a stationary code of a human being and predicting his offspring. If that code is not stationary, how can we predict changes in the person or his offspring?

Sometimes we can identify dwarfism during pregnancy. In many cases it can be identified at birth. Because of these two observations, I think it’s safe to say that dwarfism is not reversible. I’m sure you’re thinking that’s obvious, but this is coming from the guy who thinks Alzheimer’s is curable, so I need to be sure.

It’s really pretty simple [if you read my proof on Down Syndrome]: we know the age of the father is closely tied with an increased risk for dwarfism. We know that age is literally just a number, and really begins in the brain. We’ve even shown why and how this happens. And the best part: it’s reversible.

So I get it. You’re not looking to do any more research. You just want answers. You control more about the health of your baby than you know. Get healthy before you get anybody pregnant.

Sources:

Don't Read This Blog

unproven ideas that will never change the world

Category: Disease

Decrypting Diabetes [Part I]

Rethinking HIV

Cancer is a brain disease

Huntington’s is not genetic

High blood pressure starts in the brain

Tourette’s is reversible

Dads cause Dwarfism